Plasma thrombospondin-1 is increased during acute sickle cell vaso-occlusive events and associated with acute chest syndrome, hydroxyurea therapy, and lower hemolytic rates

Authors

Enrico M. Novelli, University of Pittsburgh Medical Center
Gregory J. Kato, National Heart, Lung, and Blood Institute (NHLBI)
Margaret V. Ragni, University of Pittsburgh
Yingze Zhang, University of Pittsburgh
Mariana E. Hildesheim, University of Pittsburgh
Mehdi Nouraie, The Center For Sickle Cell Disease
Suchitra Barge, University of Pittsburgh
Michael P. Meyer, ITXM-Diagnostics
Andrea Cortese Hassett, ITXM-Diagnostics
Victor R. Gordeuk, The Center For Sickle Cell Disease
Mark T. Gladwin, University of Pittsburgh
Jeffrey S. Isenberg, University of Pittsburgh

Document Type

Letter to the Editor

Publication Date

3-1-2012

Recommended Citation

Novelli, Enrico M.; Kato, Gregory J.; Ragni, Margaret V.; Zhang, Yingze; Hildesheim, Mariana E.; Nouraie, Mehdi; Barge, Suchitra; Meyer, Michael P.; Hassett, Andrea Cortese; Gordeuk, Victor R.; Gladwin, Mark T.; and Isenberg, Jeffrey S., "Plasma thrombospondin-1 is increased during acute sickle cell vaso-occlusive events and associated with acute chest syndrome, hydroxyurea therapy, and lower hemolytic rates" (2012). The Center For Sickle Cell Disease Faculty Publications. 91.
https://dh.howard.edu/sicklecell_fac/91